Anneliese Kraft*
Alzheimer’s disease (AD) is a complex neurodegenerative disorder characterized by cognitive decline, memory loss, and the accumulation of amyloid-beta plaques and tau tangles. Emerging evidence highlights the critical role of neuroinflammation in the pathogenesis of AD. Neuroinflammation, primarily mediated by glial cells, can exacerbate neuronal damage and contribute to the progression of the disease. This article reviews the current understanding of neuroinflammation in Alzheimer’s disease, including its mechanisms, the involvement of various cell types, and the potential therapeutic implications of targeting neuroinflammatory pathways.
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